I was seated
next to a type 2 diabetic acquaintance at a fundraiser church supper last
weekend. As the plates of food were passed around, I took a nice portion of ham
and then (guiltily) a serving of cole slaw. I knew it was loaded with added
sugar, but I also knew my other choices would be limited. I passed on the
scalloped potatoes au gratin and the peas, and the bread basket, and the
sweetened ice tea, each time passing the dish to my friend, who took a regular
portion of each.
I didn’t say
anything, but I thought to myself how can he do this to himself? This man is
somewhere in his eighties, skinny as a rail and looks very healthy. Maybe the
question, however, should have been, how does he get away with eating like
this? So, somehow I managed to open the subject with him. He responded by
saying, “I can eat whatever I want.” He then reached down to his belt and, out
of a small pouch, raised his pump controller to show me how he does it. My
friend, it turns out, is an insulin-dependent type 2 wearing a pump that allows
him to set the amount of basal and mealtime insulin (or “bolus”) before each
meal. So, he knew what was for supper – it’s the same every year – and he had
given himself a “shot” via the needle imbedded under his skin. Voila! He can
now “eat whatever he wants” and “cover” it with insulin.
Managing type
2 diabetes with injected insulin has heretofore always been the last resort of
pharmacotherapy. Type 2s used to be started on a course of oral pharmaceuticals
and told to continue to eat a “balanced diet.” As one drug failed to achieve
the desired control (A1c’s within the ADA recommended range of 7.0%), the dose
was increased and/or another class of oral antidiabetic drug was added until
the patient, still eating “a balanced diet,” was maxed out on three classes.
Then, the
dreaded insulin injection, by needle and later by pen, was employed for basal
(slow acting for 24 hour control) and mealtime “boluses.” Today, the introduction
of other classes of orals, and the GLP-1 injectables (Byetta, Victoza and the
once-a-week Bydureon), and most recently new drugs
(flozins) that work on
eliminating sugar in the blood via the kidneys, have enabled some patients to
delay multiple daily injections. And “the pump” has replaced
them all, for those who use it.
Another recent
development has been the introduction of insulin as a first course of treatment
(after the oral Metformin). The rationale
is that if you eat a low carb dietary, and inject a low dose of basal insulin
once or twice a day, you can potentially achieve better control by reducing
postprandial spikes (elevated blood sugars after meals), thus achieving A1c’s
in the 5s or even 4s vs.7.0%. Better control
means the surviving beta cells of the pancreas get a rest. An A1c of 7.0% is equivalent to an estimated average glucose (eAG) of
154mg/dl, but an A1c of 6 is an eAG of 126 and an A1c of 5 is an eAG of 97. This
improvement will surely reduce the possibility of the complications of poorly
controlled diabetes: retinopathy, neuropathy, and nephropathy. Translation:
blindness, amputations and end-stage kidney disease. Plus a much greater chance
of a heart
attack. Any time
your blood sugar is above 140, you are causing damage, and an average of 154
means it is above 140 a lot. It is, in my opinion, bordering on criminal to
counsel patients to only strive to achieve an A1c of 7.0%.
I don’t write
about insulin dependent type 2s because I know very little about it. Years ago
I read Richard K. Bernstein’s Diabetes Solution, the definitive source book on the subject
(for both type 1s and type 2s), but promptly forgot most of the details. His
latest edition is very highly
regarded among the cognoscenti. It is the “bible” for the growing numbers of T2s as well as T1
diabetics who have discovered the best
way to manage and control their disease is to eat Low Carb with an insulin
regimen. It is the blue print for anyone interested
in or required to inject insulin to control their diabetes.
But because I
rely on my dietary choices to directly limit
the response of my (broken) blood sugar metabolism, I’ll bet my blood sugar is
more stable, with fewer excursions, and therefore better controlled, than my
friend’s, except at this particular supper. I’d even venture to wager that my
A1c is lower than his. I didn’t ask him his, as A1c’s can
rise with age anyway. He,
and his doctor apparently, are happy with whatever it is, and he was very happy
to “eat whatever he wants.” That is another way to go. Not mine, but it
apparently works for him.
My friend was not among
these people who “eat low carb with an insulin regimen,” and he chose to “eat
whatever he wants” at the church supper. In this instance he had the advantage
over me, and I admit to being a bit jealous. If he calculated his carbs
accurately (a very big “if”), his blood sugar excursion would be
lower than mine since I had no counter
measure to employ to control mine
other than “passing” on the cole
slaw, and the strawberry shortcake for dessert. I succumbed there as well, by
the way. Alas, under some circumstances, eating Very Low Carb can be difficult and
frustrating.
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